Gordon Setter Health & Colour
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Late onset PRA in Gordon Setters
PRA (progressive retinal atrophy) refers to a group of inheritable eye diseases which affect the retina,there is no cure available at the present time and affected dogs will suffer progressive loss of vision eventually leading to total blindness. Many breeds of dog are predisposed to this condition, but the age of onset can vary, some breeds will become affected at a very early age whereas others may not develop symptoms until middle age or sometimes even older. The cause of PRA is an autosomal recessive mutation, therefore a dog must acquire two copies of the mutation, one from its sire and one from its dam to become AFFECTED. If a dog has only one copy of the mutation and one normal copy of DNA it will be a CARRIER, which means that although the dog itself won't develop PRA during its lifetime, it will pass the mutation to approximately 50% of its offspring. A dog with two normal copies of DNA is CLEAR and will not develop PRA nor pass it on to its offspring.
Tragically late onset PRA has recently been discovered in Gordon Setters in the U.K. and the U.S.A., but the Animal Health Trust in the U.K. has identified the mutation (which they have named rcd4) involved in most cases of PRA in the breed and is now offering a DNA test. The test involves a mouth swab and can be purchased from their website http://www.aht.org.uk/ at a cost of 40 GBP. A number of carriers have been identified in Australia and the Gordon Setter Clubs of Victoria and NSW strongly recommend that all breeding stock be tested for this disease prior to mating. Because of the seriousness of the disease and its potential to spread throughout the breed here in Australia, with effect from the 1st September, the Clubs will not endorse any litter or agree to advertise any litter on their websites or via their newletters unless both parents have been DNA tested (dogs proven clear by parentage excepted). This is in addition to the current requirements that both parents be x-rayed for hip dysplasia.
Source: Gordon Setter Club of Victoria
Hip (and Elbow) Dysplasia
Hip Dysplasia (HD) is the malformation of the development of one or both ball and socket joints in the hip. The hip joint is composed of the socket, which is formed by the bones of the pelvis, and the "ball" (head) of the thigh bone (femur). Normally, this joint is very tight fitting, however if suffering from dysplasia there will be too much movement in the joint resulting in wear of the joint resulting in pain and lameness. The degree of hip dysplasia present is indicated by a score assigned to each hip. Using the BVA/AVA system the hip score is the sum of the points awarded for each of nine aspects of the X-rays of both hip joints. The minimum hip score is 0 and the maximum is 106 (53 for each hip). The lower the score the less the degree of hip dysplasia present. In the UK an average (or mean) score is calculated for each breed scored under the scheme and advice for breeders is to use only breeding stock with scores well below the breed mean score.
Treatment of HD is directed at the alleviation of pain, and in severe cases involves major (and expensive) surgery to replace the joint.
Source: Gordon Setter Club of NSW
Gastric Dilation Volvulus (GDV) - More commonly known as "Bloat"
BLOAT IS AN EMERGENCY - The technical name for bloat is "Gastric Dilatation-Volvulus" ("GDV"). Bloating of the stomach is often related to swallowed air (although food and fluid can also be present). It usually happens when there's an abnormal accumulation of air, fluid, and/or foam in the stomach ("gastric dilatation"). Stress can be a significant contributing factor also. Bloat can occur with or without "volvulus" (twisting). As the stomach swells, it may rotate 90Â° to 360Â°, twisting between its fixed attachments at the esophagus (food tube) and at the duodenum (the upper intestine). The twisting stomach traps air, food, and water in the stomach. The bloated stomach obstructs veins in the abdomen, leading to low blood pressure, shock, and damage to internal organs. The combined effect can quickly kill a dog. It is frequently reported that deep-chested dogs, such as German Shepherds, Great Danes, and Dobermanns are particularly at risk.
Please note that Liver or Red coat colours in Gordon Setters, is not a health issue!
What is the effect of recessive colour genes in the Gordon Setter? If a black and tan (b&t) Gordon who is a carrier of the recessive red gene is mated to another b&t Gordon who is also a carrier of the recessive red gene then about 25% of the resulting litter will be red instead of b&t. So in a litter of 8 puppies you would probably have about 2 red puppies. If a carrier for red is mated to a non carrier for red then all the puppies will be b&t but about 50% will be carriers of the recessive red gene. In the first example where both parent are carriers of the recessive red gene about 66% of the b&t puppies will be carriers of the recessive red gene. In other words in our litter of 8, on average 2 will be red, 4 will be b&t but carrying the recessive red gene and 2 will be b&t and not carrying the recessive red gene. (Please note the use of terms such as 'about' and 'on average'. The inheritance of genetic characteristics is the probability of an event happening based on large sample sizes and the actual percentages can vary considerably if the sample size is small.)
Source: Gordon Setter Club of NSW
Steve, Carolyn & Laura Bennett & Sarah Auld
Macclesfield, Adelaide Hills, 5153, SA, Australia
Phone : SB0414930223-CB0419839259-SA0407653697
Email : [email protected]